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  3. Vol. 23 No. 04 (2026): July-August 2026
  4. ORIGINAL PAPER(UROLOGICAL ONCOLOGY)

Vol. 23 No. 04 (2026)

September 2026

Timing of Radiographic Progression Relative to Prostate-Specific Antigen Kinetics in Patients with Metastatic Castration-Sensitive Prostate Cancer Receiving Upfront Androgen Receptor Signaling Inhibitor Therapy

  • Akinori Nakayama
  • Erika Ikezoe
  • Minoru Inoue
  • Hiroki Tsujioka
  • Asumi Nirazuka
  • Keita Izumi
  • Kiyoshi Setoguchi
  • Kazutaka Saito

Urology Journal, Vol. 23 No. 04 (2026), 20 September 2026 , Page 167-171
https://doi.org/10.22037/uj.v23i04.8771 Published: 2026-09-20

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Abstract

Purpose: Prostate-specific antigen (PSA) progression does not always coincide with radiographic progression in
patients with metastatic castration-sensitive prostate cancer (mCSPC) receiving upfront androgen receptor signaling inhibitor (ARSI) therapy. We evaluated the timing of radiographic progression according to predefined PSA
kinetic periods.
Materials and Methods: We retrospectively reviewed 82 consecutive patients with synchronous mCSPC treated
with upfront ARSI therapy between April 2018 and May 2024. The median follow-up duration was 23 months
(interquartile range [IQR], 14–38). PSA kinetics were categorized into three predefined periods: pre-PSA nadir
(treatment initiation to PSA nadir), post-PSA nadir/before PSA progression (PSA nadir to PSA progression), and
post-PSA progression. Radiographic progression events were classified according to these PSA kinetic periods.
Results: During follow-up, 33 patients (40%) experienced PSA progression and 26 (31.7%) developed radiographic
progression. Among the 26 radiographic progression events, 1 (3.8%) occurred during the pre-PSA nadir
period, 7 (26.9%) during the post-PSA nadir/before PSA progression period, and 18 (69.2%) during the post-
PSA progression period. Overall, 25 of 26 radiographic progression events (96.2%; 95% confidence interval [CI]:
81.1%–99.3%) occurred after PSA nadir. The only patient who developed radiographic progression before PSA
nadir was subsequently diagnosed with small-cell carcinoma of the prostate. PSA nadir levels and time to PSA nadir
were comparable between patients who developed radiographic progression before and after PSA progression.
Conclusion: Radiographic progression before PSA nadir was uncommon in this cohort, whereas a substantial
proportion of progression events occurred after PSA nadir but before PSA progression. These findings suggest
that PSA nadir may serve as a useful temporal reference point for radiographic surveillance during upfront ARSI
therapy. However, these observations should be considered hypothesis-generating and require validation in larger
prospective multicenter studies.

Keywords:
  • androgen receptor signaling inhibitors
  • metastatic castration-sensitive prostate cancer
  • prostate-specific antigen kinetics
  • radiographic progression
  • upfront therapy
  • 8771/pdf

How to Cite

Nakayama, A., Ikezoe, E., Inoue, M., Tsujioka, H., Nirazuka, A., Izumi, K., … Saito, K. (2026). Timing of Radiographic Progression Relative to Prostate-Specific Antigen Kinetics in Patients with Metastatic Castration-Sensitive Prostate Cancer Receiving Upfront Androgen Receptor Signaling Inhibitor Therapy. Urology Journal, 23(04), 167–171. https://doi.org/10.22037/uj.v23i04.8771
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References

Armstrong AJ, Szmulewitz RZ, Petrylak DP, Holzbeierlein J, Villers A, Azad A, et al. ARCHES: A randomized, phase III study of androgen deprivation therapy with enzalutamide or placebo in men with metastatic hormone-sensitive prostate cancer. J Clin Oncol. 2019;37:2974-86.

Chi KN, Agarwal N, Bjartell A, Chung BH, Pereira de Santana Gomes AJ, Given R, et al. Apalutamide for metastatic, castration-sensitive prostate cancer. N Engl J Med. 2019;381:13-24.

Fukuokaya W, Yanagisawa T, Mori K, Katayama S, Fukuda H, Uchimoto T, et al. Radiographic progression without corresponding prostate-specific antigen progression in patients with metastatic castration-sensitive prostate cancer receiving apalutamide: secondary analysis of the Titan trial. Eur Urol Oncol. 2024;S2588-9311(24)00101-9.

Armstrong AJ, Mottet N, Iguchi T, Shinohara N, Zorn KC, Weitzman AL, et al. Radiographic progression in the absence of prostate-specific antigen (PSA) progression in patients with metastatic hormone-sensitive prostate cancer (mHSPC): post hoc analysis of ARCHES. J Clin Oncol. 2022;40(16_suppl):5072.

Guerrero J, Alfaro IE, Gómez F, Protter AA, Bernales S. Enzalutamide, an androgen receptor signaling inhibitor, induces tumor regression in a mouse model of castration-resistant prostate cancer. Prostate. 2013;73:1291-305.

Gillessen S, Bossi A, Davis ID, Saad F, Miller K, Attard G, et al. Management of patients with advanced prostate cancer-metastatic and/or castration-resistant prostate cancer: report of the Advanced Prostate Cancer Consensus Conference (APCCC) 2022. Eur J Cancer. 2023;185:178-215.

Scher HI, Morris MJ, Stadler WM, Higano CS, Basch E, Fizazi K, et al. Trial design and objectives for castration-resistant prostate cancer: updated recommendations from the Prostate Cancer Clinical Trials Working Group 3. J Clin Oncol. 2016;34:1402-18.

Eisenhauer EA, Therasse P, Bogaerts J, Schwartz LH, Sargent D, Ford R, et al. New response evaluation criteria in solid tumours: Revised RECIST guideline (version 1.1). Eur J Cancer. 2009;45:228-47.

Yamada Y, Beltran H. Clinical and biological features of neuroendocrine prostate cancer. Curr Oncol Rep. 2021;23:15.

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