School of Medicine Students' Journal
  • Register
  • Login
  • Home
  • Issues
    • Current
    • Archives
  • Journal Info
    • About the Journal
    • Aims and Scope
    • Editorial Team
    • Privacy Statement
    • Contact
  • Guidelines
    • Authors' Guideline
    • Forms
  • Publication Ethics
  • Contact Us
  • Announcements
  • Submit
Advanced Search
  1. Home
  2. Archives
  3. Vol. 1 No. 1 (2019)
  4. Case Report

Vol. 1 No. 1 (2019)

Shock Following Percutaneous Coronary Intervention as a Manifestation of Diabetes Insipidus: A Case Report

  • Younes Nozari
  • Abbas Andishmand
  • Akbar Shafiee

School of Medicine Students' Journal, Vol. 1 No. 1 (2019), , Page 21-23
https://doi.org/10.22037/smsj.v1i1.15791 Published 20 May 2018

  • View Article
  • Download
  • Cite
  • References
  • Statastics
  • Share

Abstract

The shock syndrome following percutaneous coronary intervention (PCI) is a life-threatening complication, accompanied by poor outcomes. The most common causes are bleeding and severe left ventricular systolic dysfunction; albeit rare complications, they are still challenging. We herein report a 47-year-old man who suffered from complications following PCI, manifested as unexplained shock, which was resistant to conventional management.

Overestimating the role of the patient’s underlying disorder as the cause of the hypotension led to delay in diagnosis and treatment; however, obtaining a thorough familial history revealed diabetes insipidus (DI) as the most probable cause of the hypotension despite normal electrolyte levels at the time of admission. The patient dramatically responded to DI management.  

Persistent hypotension or shock after PCI can result from an unusual or rare etiology. Meticulous history taking and attention to clinical findings are essential for an accurate diagnosis and timely treatment of this life-threatening condition.

Keywords:
  • Shock syndrome
  • Diabetes insipidus
  • percutaneous coronary intervention
  • PDF

How to Cite

1.
Nozari Y, Andishmand A, Shafiee A. Shock Following Percutaneous Coronary Intervention as a Manifestation of Diabetes Insipidus: A Case Report. Sch Med Stud J [Internet]. 2018May20 [cited 2022May25];1(1):21-3. Available from: https://journals.sbmu.ac.ir/smsj/article/view/15791
  • ACM
  • ACS
  • APA
  • ABNT
  • Chicago
  • Harvard
  • IEEE
  • MLA
  • Turabian
  • Vancouver
  • Endnote/Zotero/Mendeley (RIS)
  • BibTeX

References

S. Valente, C. Lazzeri, S. Vecchio, C. Giglioli, M. Margheri, P. Bernardo, et al.Predictors of in-hospital mortality after percutaneous coronary intervention for cardiogenic shock.International journal of cardiology.2007;114(2):176-82.

Eng MH,Moses JW,Teirstein PS.Complications of Percutaneous Coronary Intervention. In: Topol EJ. Textbook of Interventional Cardiology.Elsevier. 6th edition. pp 357-371, 2012.

G. Dangas, I. Iakovou, E. Nikolsky, E. D. Aymong, G. S. Mintz, N. N. Kipshidze, et al.Contrast-induced nephropathy after percutaneous coronary interventions in relation to chronic kidney disease and hemodynamic variables.The American journal of cardiology.2005;95(1):13-19.

Ragosta M .Retroperitoneal bleeding.In:Cases in Interventional Cardiology. Elsevier –Saunders ,pp 135-8, 2011.

J. Bakker, P. Gris, M. Coffernils, R. J. Kahn and J. L. Vincent.Serial blood lactate levels can predict the development of multiple organ failure following septic shock.The American journal of surgery.1996;171(2):221-26.

M. H. Weil, J. Leavy and E. C. Rackow.Prognosis in shock.Anaesthesia.1986;41(1):80-2.

A. Meregalli, R. P. Oliveira and G. Friedman.Occult hypoperfusion is associated with increased mortality in hemodynamically stable, high-risk, surgical patients.Crit Care.2004;8(2):R60-5.

Ragosta M.Complications of cardiac catheterization .In:Cardiac Catheterization. Elsevier –Saunders ,pp 21-31,2010.

A. N. Makaryus and S. I. McFarlane.Diabetes insipidus: diagnosis and treatment of a complex disease.Cleveland Clinic journal of medicine.2006;73(1):65-71.

F. Rauch, C. Lenzner, P. Nürnberg, C. Frömmel and U. Vetter.A novel mutation in the coding region for neurophysin‐II is associated with autosomal dominant neurohypophyseal diabetes insipidus.Clinical endocrinology.1996;44(1):45-51.

D. R. Repaske, R. Medlej, E. K. Gültekin, M. Krishnamani, G. Halaby, J. W. Findling, et al.Heterogeneity in clinical manifestation of autosomal dominant neurohypophyseal diabetes insipidus caused by a mutation encoding Ala− 1→ Val in the signal peptide of the arginine vasopressin/neurophysin II/copeptin precursor.Journal of Clinical Endocrinology & Metabolism.1997;82(1):51-56.

  • Abstract Viewed: 2491 times
  • PDF Downloaded: 93 times

Download Statastics

  • Linkedin
  • Twitter
  • Facebook
  • Google Plus
  • Telegram
Make a Submission
ALT ALT ALT
Browse
  • Categories
    • SMSJ Peds.
    • Covid-19
    • Visual Practice
Keywords
Information
  • For Readers
  • For Authors
Current Issue
  • Atom logo
  • RSS2 logo
  • RSS1 logo
  • Home
  • Archives
  • Submissions
  • About the Journal
  • Editorial Team
  • Contact

This journal is licensed under a Creative Commons Attribution 4.0 International License.

eISSN: 2676-7597

The template of this website is designed by Sinaweb